By Neil D. Kitchen, Guy McKhann, Hadi Manji
This illustrated color evaluation covers all elements of neurology and neurosurgery together with: dystonia, tremor, akinetic inflexible syndrome (Parkinsonian conditions), infectious illnesses, headache, mind tumors, demyelinating ailment, epilepsy, neuro-ophthalmology, peripheral neuropathy, medical neurophysiology, pituitary, coma, neurogenetics, surgical process, hydrocephalus, AVM/aneurysm, soreness and trigeminal neuralgia, head damage, spinal damage, stroke and neuroradiology. The editors and contributing authors all specialize in neurology and/or neurosurgery. The publication is geared toward execs in pr. Read more...
summary: This illustrated color evaluation covers all elements of neurology and neurosurgery together with: dystonia, tremor, akinetic inflexible syndrome (Parkinsonian conditions), infectious illnesses, headache, mind tumors, demyelinating affliction, epilepsy, neuro-ophthalmology, peripheral neuropathy, scientific neurophysiology, pituitary, coma, neurogenetics, surgical strategy, hydrocephalus, AVM/aneurysm, discomfort and trigeminal neuralgia, head damage, spinal damage, stroke and neuroradiology. The editors and contributing authors all concentrate on neurology and/or neurosurgery. The booklet is aimed toward pros in pr
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Extra info for Clinical Neurology and Neurosurgery : Self-Assessment Colour Review
Iii. The causes of a pure motor syndrome causing wasting of the small hand muscles are few – either a neuronopathy or a motor neuropathy. In this context, MND is the most likely neuronopathy. The causes of a pure motor neuropathy are also limited and usually one of the following: MMNCB/CIDP, lead neuropathy, or neuropathy secondary to acute intermittent porphyria. The investigations are as follows: for MND, neurophysiology (normal NCS with denervation on needle EMG); for MMNCB, neurophysiology (evidence of conduction block on NCS which may be proximal); for lead poisoning, 24-hour urinary lead; for acute intermittent porphyria, urinary urobilinogen and porphobilinogen.
After elaborating on the associated surgical risks of the plethora of available procedures, the patient is adamant in receiving the most effective and long-lasting treatment. Which procedure would you recommend? 57 48, 49: Answers 48 i. The resting CMAP amplitude is markedly reduced. e. after exercise) potentiation of >200%. This indicates a pre-synaptic neuromuscular transmission disorder. ii. These findings are indicative of the LEMS. iii. Repetitive nerve stimulation at slow rates (1–3/s) elicits significant decrement.
5 mg/dl). i. What abnormality is shown on the MRI scan (19)? ii. What other non-neurologic investigation may be necessary? iii. Are there any other tests that can be performed on the CSF? iv. What is the treatment? 19 29 18, 19: Answers 18 i. The images are T1-weighted (18a) and T2-weighted (18b) sequences using the spin-echo technique. On a T1-weighted image, gray matter is lower in signal intensity than white matter, fat is bright, and CSF is dark. On a true T2-weighted image, gray matter is higher in signal intensity than white matter, CSF is bright and fat is dark.