By Gloria J Hathaway; Nick H Proctor; James P Hughes
ISBN-10: 0471268836
ISBN-13: 9780471268833
Read Online or Download Proctor & Hughes' chemical hazards of the workplace PDF
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Sample text
Gagnaire F, Zissn D, Bonnet P, et al: Nasal and pulmonary toxicity of allyl glycidyl ether in mice. Toxicol Lett 39:139–145, 1987 5. National Toxicology Program. Toxicology and Carcinogenesis Studies of Allyl Glycidyl Ether (CAS No. 106-92-3) in Osborne Mendel Rats and B6C3F1 Mice (Inhalation Studies). Technical Report No. 376. Public Health Service, National Institutes of Health. NIH Pub. No. 90-2831, Research Triangle Park, NC, 1990 6. Renne RA, Brown HR, Jokinen MP: Morphology of nasal lesions induced in OsborneMendel rats and B6C3F1 mice by chronic inhalation of allyl glycidyl ether.
Starting material for acrylates and polyacrylates used in plastics, water purification, paper and cloth coatings, and medical and dental materials Exposure. Inhalation; ingestion Toxicology. Acrylic acid is a severe irritant of the eyes, nose, and skin. The major route of absorption is ingestion of inhaled vapors. 1 Although acrylic acid is acutely irritating at sites of initial contact, it causes little systemic toxicity. 6 At 300 ppm, twenty 6-hour exposures produced all but the latter effect.
Colorless liquid Uses. Manufacture of acrylic fibers; synthesis of rubberlike materials; pesticide fumigant Exposure. Inhalation; skin absorption Toxicology. Acrylonitrile is an eye, skin, and upper respiratory tract irritant; systemic effects are nonspecific but may include the central nervous, hepatic, renal, cardiovascular, and gastrointestinal systems. It is carcinogenic in experimental animals. 1 Acrylonitrile is metabolized to cyanide by hepatic microsomal reactions. Deaths from acute poisoning result from inhibition of mitochondrial cytochrome oxidase activity by metabolically liberated cyanide.